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MOTS-c

Mitochondrial-Derived Signaling Peptide

Best For

Metabolic optimization, mitochondrial support, exercise recovery and performance

16aa

Amino acid peptide encoded in mitochondrial DNA

AMPK

Master metabolic switch activated by MOTS-c

40%

Improvement in glucose tolerance in preclinical models

How It Works

MOTS-c is a 16-amino-acid peptide encoded within the mitochondrial 12S rRNA gene — one of the first identified mitochondrial-derived peptides with systemic signaling activity. It activates AMPK (AMP-activated protein kinase) in skeletal muscle and adipose tissue, mimicking the metabolic effects of exercise by enhancing glucose uptake and fatty acid oxidation. MOTS-c also regulates the folate-methionine cycle, shifting cellular metabolism toward improved NAD+ biosynthesis and reducing inflammatory cytokine expression. Research from the Lee Lab at USC has demonstrated that MOTS-c translocates to the nucleus under metabolic stress, directly regulating gene expression related to cellular resilience.

Key Research Areas

Exercise Mimetic

Activates AMPK pathways that mirror the metabolic benefits of physical exercise

Insulin Sensitivity

Improves glucose uptake in skeletal muscle independent of insulin signaling

Mitochondrial Function

Enhances NAD+ biosynthesis and mitochondrial oxidative capacity

Metabolic Inflammation

Reduces TNF-alpha and IL-6 expression in adipose and muscle tissue

Research Highlights

  • First mitochondrial-derived peptide shown to translocate to the nucleus and regulate gene expression
  • Identified by the Lee Lab at USC — a leading center for mitochondrial peptide research

Formulation Notes

Common concentrations referenced in published clinical studies:

[MS10]10mg[MS40]40mg

Category

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Educational Content. This compound profile is for informational purposes only. Not intended as medical advice, diagnosis, or treatment.

Evidence reviewed: March 2026